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Matthiesen S, Kempkens S, Bahulayan A, Juergens UR, Racke K
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N. M”sslein1, D. Zindel1,2, J. K. S. Tiemann3, M. E. Sommer4, J. Pin2,5, L. Pr‚zeau2,5, P. W. Hildebrand3, M. Bnemann1, C. Krasel1. 1Institute of Pharmacology and Clinical Pharmacy, University of Marburg, Marburg, Germany, 2Institut de G‚nomique Fonctionnelle (IGF), University of Montpellier, Montpellier, France, 3Institute for Medical Physics and Biophysics, University of Leipzig, Leipzig, Germany, 4Institute for Medical Physics and Biophysics, Charit‚ Berlin, Berlin, Germany, 5INSERM U1191, Montpellier, France,
- Mutations in the S1P1 binding pocket differentially affect S1P and FTY720-P-induced signalling
van Loenen PB, de Graaf C, Verzijl D, Leurs R, Rognan D, Peters SLM, Alewijnse AE
- Mutations within the M3-M4 cytoplasmic loop significantly reduce inward rectification in human 5-HT3A receptors
S.P. Kelley; J.I. Dunlop; E.F. Kirkness; J.J. Lambert; J.A. Peters
- MYOCARDIAL POSTCONDITIONING IS MIMICKED BY B-TYPE NATRIURETIC PEPTIDE BUT NOT EXOGENOUS NITRIC OXIDE
Dwaine S BurleyGary F Baxter
- Myocardial protection with low-molecular dinitrosyl iron complexes
Pisarenko Oleg, Serebryakova Larisa, Studneva Irina, Shulzhenko Valentin, Tskitishvili Olga, Timoshin Alexander, Vanin Anatoly
- MYOCARDIUM ENZYME ACTIVITIES IN CHOLINE-DEFICIENT ADULT RATS: THE ROLE OF CARNITINE
Strilakou A, Tsakiris S, Kalafatakis K, Stylianaki A, Perelas A, Koulouris A, Liapi C
- N-Acylethanolamine-hydrolizing Acid Amidase (NAAA) in the EAE Model of Multiple Sclerosis
Capurro V, Pontis S, Dionisi M, Busquet P, Bertorelli R, Tarozzo G, Reggiani A, Piomelli D
- NADPH oxidase 1 and 4 are relevant sources of oxidative stress and therapeutic targets in hypertension and stroke
Schmidt H, Hermann R, Schiffers P, Lam E, Ho H, Vo L, Kleinschnitz C, Wingler K
- Na+/H+ exchanger isoform 1-induced osteopontin expression facilitates cardiomyocyte hypertrophy through CD44
F Mraiche, N Abdulrahman, L Fliegel, A Gadeau
